유키오 야마모토, Yukio Yamamoto, Ph.D.

Principal Researcher

OFFICE

L7204, +82-2-958-7223

LAB

L7215, +82-2-958-7223

EDUCATION

Ph.D. Graduate School of Veterinary Medicine, Hokkaido University (1998)

B.S. Faculty of Veterinary Medicine, Hokkaido University (1994)

RESEARCH CAREER & ACTIVITIES
  • Principal Researcher, Center for Functional Connectomics, Brain Science Institute, Korea Institute of Science and Technology (KIST) (2018-Present)
  • Research Scientist, Center for Functional Connectomics, Brain Science Institute, Korea Institute of Science and Technology (KIST) (2010-2018)
  • Research Associate Professor in MTT Program, Medical Research Institute, Tokyo Medical and Dental University (2007-2010)
  • Research Fellow, National Institute of Environmental Health Sciences, National Institutes of Health (2006-2007)
  • Visiting Fellow, National Institute of Environmental Health Sciences, National Institutes of Health (2001-2006)
  • Postdoctoral Research Fellow, The 4th Department, Osaka Bioscience Institute (1998-2001)
RESEARCH INTERESTS
  • Developing molecular tools for targeting specific neuronal cell type
  • Developing optogenetic molecules to regulate cell signaling
  • Elucidating neuronal network dependent gene expression
HIGHLIGHT
  •  Kim T, Yamamoto Y*, Tanaka-Yamamoto K*. Timely regulated sorting from early to late endosomes is required to maintain cerebellar long-term depression. Nat Commun. 2017, 8(1):401.
  • Lee D, Yamamoto Y, Kim E, Tanaka-Yamamoto K. Functional and Physical Interaction of Diacylglycerol Kinase ζ with Protein Kinase Cα Is Required for Cerebellar Long-Term Depression. J Neurosci. 2015, 35(46):15453-65.
  • Kim Y, Kim T, Rhee JK, Lee D, Tanaka-Yamamoto K*, Yamamoto Y*. Selective transgene expression in cerebellar Purkinje cells and granule cells using adeno-associated viruses together with specific promoters. Brain Res. 2015, 1620:1-16.
  • Yamamoto Y, Lee D, Kim Y, Lee B, Seo C, Kawasaki H, Kuroda S, Tanaka-Yamamoto K. Raf kinase inhibitory protein is required for cerebellar long-term synaptic depression by mediating PKC-dependent MAPK activation. J Neurosci. 2012, 32(41):14254-64.